ScienceDaily: Top Health News |
- Flipping the 'off' switch on cell growth: Protein uses multiple means to help cells cope when oxygen runs low
- Race linked to childhood food allergies, not environmental allergies
- Reprogramming cells to fight diabetes
- Head and neck cancer molecular tumor subtypes documented
| Posted: 23 Feb 2013 08:15 AM PST A protein known for turning on genes to help cells survive low-oxygen conditions also slows down the copying of new DNA strands, thus shutting down the growth of new cells, researchers report. Their discovery has wide-ranging implications, they say, given the importance of this copying -- known as DNA replication -- and new cell growth to many of the body's functions and in such diseases as cancer. |
| Race linked to childhood food allergies, not environmental allergies Posted: 23 Feb 2013 08:15 AM PST Researchers have shown that race and possibly genetics play a role in children's sensitivity to developing allergies. Researchers found: African-American children were sensitized to at least one food allergen three times more often than Caucasian children. African-American children with one allergic parent were sensitized to an environmental allergen twice as often as African-American children without an allergic parent. |
| Reprogramming cells to fight diabetes Posted: 23 Feb 2013 08:13 AM PST For years researchers have been searching for a way to treat diabetics by reactivating their insulin-producing beta cells, with limited success. The "reprogramming" of related alpha cells into beta cells may one day offer a novel and complementary approach for treating type 2 diabetes. Treating human and mouse cells with compounds that modify cell nuclear material called chromatin induced the expression of beta cell genes in alpha cells, according to a new study. |
| Head and neck cancer molecular tumor subtypes documented Posted: 23 Feb 2013 08:13 AM PST By analyzing data from DNA microarrays, scientists have completed a study that confirms the presence of four molecular classes of the disease and extends previous results by suggesting that there may be an underlying connection between the molecular classes and observed genomic events, some of which affect known cancer genes. |
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